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The inflammatory microenvironment by which SARMs can affect immune function is unknown since it is unclear how the microenvironment of tissue changes following SARMs administration. In rodents, the tissue microenvironment is changed upon administration of testosterone and other SARMs. As demonstrated by our human study, the level of neutrophils, monocytes, and lymphocytes increased after treatment of prostate cells with 1,5-methyl-4-isoxazolepropionic acid (DMPA) (11) and testosterone (12), and serum concentrations of TNFα, IL-6, and MCP-1 were higher than those without testosterone in the prostate tissue from men treated with DMPA (12). Further, 1,5-methyl-4-isoxazolepropionic acid induces expression of the inflammatory cytokine MCP-1 in cultured monocytes via up-regulation of genes encoding TNFα and IκB
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